Search results for "ISM [Radio continuum]"

showing 10 items of 703 documents

Neuroblastoma after Childhood: Prognostic Relevance of Segmental Chromosome Aberrations, ATRX Protein Status, and Immune Cell Infiltration

2014

AbstractNeuroblastoma (NB) is a common malignancy in children but rarely occurs during adolescence or adulthood. This subgroup is characterized by an indolent disease course, almost uniformly fatal, yet little is known about the biologic characteristics. The aim of this study was to identify differential features regarding DNA copy number alterations, α-thalassemia/mental retardation syndrome X-linked (ATRX) protein expression, and the presence of tumor-associated inflammatory cells. Thirty-one NB patients older than 10 years who were included in the Spanish NB Registry were considered for the current study; seven young and middle-aged adult patients (range 18-60 years) formed part of the c…

MaleCancer ResearchHet heterogeneousGene ExpressionNeuroblastomaImmunophenotypingRegistriesYoung adultNeoplasm MetastasisMLPA multiplex ligation probe amplificationChildIn Situ Hybridization FluorescenceNuclear ProteinsMiddle AgedAYA adolescent and young adultsPrognosislcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensNCA numerical chromosome aberrationImmunohistochemistryFemaleSCA segmental chromosome aberrationIHC immunohistochemistryNB neuroblastomaAdultX-linked Nuclear ProteinAdolescentaSNP single nucleotide polymorphism arrayBiologyMalignancyChromosome aberrationPolymorphism Single Nucleotidelcsh:RC254-282ArticleImmunophenotypingYoung AdultLymphocytes Tumor-InfiltratingNeuroblastomacnLOH copy-neutral loss of heterozygositymedicineHumansHom homogeneousATRXNeoplasm StagingChromosome AberrationsDNA Helicasesmedicine.diseaseSpainMNNA MYCN not amplifiedCancer researchFSCA focal segmental chromosome aberrationCD8MNA MYCN amplifiedNeoplasia
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Pyrrolotetrazinones deazaanalogues of temozolomide induce apoptosis in Jurkat cell line: involvement of tubulin polymerization inhibition.

2009

Pyrrolotetrazinones are a new class of azolotetrazinones endowed with a high, remarkable antiproliferative activity in human tumor cultured cells. They hold the deaza skeleton of the antitumor drug temozolomide, although preliminary investigations indicated a different mechanism of action. To understand their mechanism(s) of action along with their target at molecular level, four derivatives were selected on the basis of their activity on a panel of human tumor cell lines and they were investigated in depth in a T leukemia cell line (Jurkat). Flow cytometric analysis of cell cycle after treatment with pyrrolotetrazinones has demonstrated that they were able to induce an arrest of the cell c…

MaleCancer ResearchProgrammed cell deathCarcinoma HepatocellularCell SurvivalCellGene ExpressionAntineoplastic AgentsApoptosisPhosphatidylserinesBiologyToxicologyJurkat cellsMicrotubulesMicrotubule polymerizationJurkat CellsMiceTubulinCell Line TumormedicineTemozolomideAnimalsHumansPharmacology (medical)Cell Proliferationbcl-2-Associated X ProteinPharmacologyMembrane Potential MitochondrialMice Inbred BALB CCaspase 3Cell CycleCell MembraneCell cycleSettore CHIM/08 - Chimica FarmaceuticaTubulin ModulatorsCell biologyMitochondriaDacarbazinemedicine.anatomical_structureOncologyMechanism of actionBiochemistryProto-Oncogene Proteins c-bcl-2ApoptosisCell culturemedicine.symptomPoly(ADP-ribose) PolymerasesReactive Oxygen SpeciesPyrrolotetrazinoneCancer chemotherapy and pharmacology
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The tumour-inhibiting potential of the progesterone antagonist Onapristone in the human mammary carcinoma T61 in nude mice

1992

The progesterone antagonist Onapristone proved to possess strong tumour-inhibiting activity in a panel of experimental mammary carcinomas. Its underlying mechanism of action is due to a progesterone-receptor-mediated induction of terminal differentiation and a specific blockade of the cell cycle and is also present in the absence of progesterone as was shown in the MXT mammary tumour. To prove this further, the tumour-inhibiting activity of Onapristone was investigated in the human postmenopausal T61 mammary tumour implanted in castrated male nude mice. Whereas Onapristone given alone had no effect on growth of established tumours, after stimulation of the relatively low progesterone recept…

MaleCancer Researchmedicine.medical_specialtyTransplantation HeterologousMammary glandMice NudeAntineoplastic AgentsStimulationEndogenyGonanesBiologyProgesterone AntagonistMiceInternal medicineProgesterone receptormedicineAnimalsHumansReceptorProgesteroneAntagonistMammary Neoplasms ExperimentalGeneral Medicinemedicine.anatomical_structureEndocrinologyOncologyMechanism of actionFemalemedicine.symptomNeoplasm TransplantationJournal of Cancer Research and Clinical Oncology
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Noradrenaline release from permeabilized synaptosomes is inhibited by the light chain of tetanus toxin

1992

AbstractNoradrenaline release from rat brain cortical synaptosomes permeabilized with streptolysin O can be triggered by μM concentrations of free Ca2+. This process was inhibited within minutes by tetanus toxin and its isolated light chain, but not by its heavy chain. The data demonstrate that the effect of tetanus toxin on NA release from purified synaptosomes is caused by the intraterminal action of its light chain.

MaleCell Membrane PermeabilityClostridium tetaniBiophysicsBiologymedicine.disease_causeImmunoglobulin light chainBiochemistryExocytosisExocytosisGeneeskundeNorepinephrineStructural BiologyPermeabilizationGeneticsmedicineSynaptosomeAnimalsNeurotoxinRats WistarStreptolysin OMolecular BiologySynaptosomeToxinCell BiologyRatsTetanus toxinMechanism of actionBiochemistryStreptolysinmedicine.symptomSynaptosomesFEBS Letters
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Memory-Based Mismatch Response to Frequency Changes in Rats

2011

Any occasional changes in the acoustic environment are of potential importance for survival. In humans, the preattentive detection of such changes generates the mismatch negativity (MMN) component of event-related brain potentials. MMN is elicited to rare changes (‘deviants’) in a series of otherwise regularly repeating stimuli (‘standards’). Deviant stimuli are detected on the basis of a neural comparison process between the input from the current stimulus and the sensory memory trace of the standard stimuli. It is, however, unclear to what extent animals show a similar comparison process in response to auditory changes. To resolve this issue, epidural potentials were recorded above the pr…

MaleCentral Nervous SystemMismatch negativityCentral auditory processingAudiologylocal field potentials170 EthicsRats Sprague-DawleyCognitionLearning and Memory0302 clinical medicine10007 Department of Economicsratchange detectionEvoked Potentialsta515media_commonMultidisciplinarySensory memorymuutoksen havaitseminenQ05 social sciencesRAnimal ModelsNeuroethologykuuloSensory Systems330 Economicsmedicine.anatomical_structureAuditory SystemTone FrequencyEvoked Potentials AuditoryMedicineSensory PerceptionResearch ArticlePsychoacousticsmedicine.medical_specialtyScienceCognitive Neurosciencemedia_common.quotation_subjectNeurophysiologyU5 Foundations of Human Social Behavior: Altruism and Egoism1100 General Agricultural and Biological SciencesaistimuistiStimulus (physiology)sensory memoryAuditory cortexprimaarikuuloaivokuoribehavioral disciplines and activities050105 experimental psychology03 medical and health sciencesModel Organisms1300 General Biochemistry Genetics and Molecular BiologyMemoryprimary auditory cortexPerceptionPsychophysicsmedicineAnimalsAuditory system0501 psychology and cognitive sciencesBiology1000 Multidisciplinarybusiness.industryAnimal CognitionRatsrottakoe-esiintyminenRatbusiness030217 neurology & neurosurgeryNeuroscience
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Effects of Propofol on H-reflex in Humans

2001

Background Depression of spinal cord motoneuron excitability has been proposed to contribute to surgical immobility. The H-reflex, which measures alpha-motoneuron excitability, is depressed by volatile anesthetics, whereas the action of propofol is unknown. The objective of this study was to determine the effects of propofol anesthesia on the H-reflex. Methods In 13 patients (group 1), H-reflex was measured before (T0), 3 min after (T1), and 10 min after (T2) a 2-mg/kg bolus dose of propofol, followed by an infusion of 10 mg x kg(-1) x h(-1). Ten patients (group 2) were studied when propofol was given via a programmable pump set to a propofol blood concentration of 6 microg/ml, and 10 pati…

MaleCentral nervous systemH-ReflexmedicineHumansMuscle SkeletalPropofolMotor Neuronsbusiness.industryVolatile anestheticMiddle AgedSpinal cordElectric StimulationElectrophysiologyAnesthesiology and Pain Medicinemedicine.anatomical_structureMechanism of actionAction (philosophy)AnesthesiaFemaleH-reflexmedicine.symptomAnesthesia InhalationbusinessPropofolNeuroscienceAnesthetics Intravenousmedicine.drugAnesthesiology
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Interaction of uridine with GABA binding sites in cerebellar membranes of the rat

1983

The effect of uridine, a postulated anticonvulsant agent, on GABA receptors has been investigated. Uridine inhibits [3H]GABA binding to rat cerebellar buffer-washed membranes. Pretreatment of the membranes with Triton X-100 increases the effect of uridine on GABA-binding. The Scatchard analysis reveals that both high and low affinities of GABA for its receptors are affected by 1 mM uridine, while the apparent number of binding sites remains unchanged. The ability of uridine to interact competitively with GABA binding sites, also examined by the Lineweaver-Burk analysis, suggests a possible mechanism of action of this anticonvulsant agent, so including it among those compounds characterized …

MaleCerebellumReceptors Cell SurfaceBiologyBinding CompetitiveBiochemistrygamma-Aminobutyric acidCellular and Molecular Neurosciencechemistry.chemical_compoundGABA receptorCerebellummedicineAnimalsBinding siteReceptorUridinegamma-Aminobutyric AcidGABAA receptorCell MembraneRats Inbred StrainsGeneral MedicineReceptors GABA-AUridineRatsmedicine.anatomical_structurenervous systemBiochemistryMechanism of actionchemistryAnticonvulsantsmedicine.symptommedicine.drugNeurochemical Research
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Local Cerebral Blood Flow in a Rat Cortical Vein Occlusion Model

1996

The symptoms following sinus and vein occlusion observed in patients and experimental animals display a considerable variability that so far remains largely unexplained. In a rat cortical vein occlusion model using a photochemical thrombotic technique, we examined changes in the cerebral venous flow pattern by fluorescence angiography and regional cerebral blood flow (rCBF) and cerebral blood volume fraction (CBVF) by a modern laser Doppler “scanning” technique. Brain damage was assessed histologically. Fluorescence angiographic findings fell into two groups: group A, rats with an altered venous flow pattern after occlusion (n = 12), and group B, rats with interruption of blood flow and/or…

MaleCerebral veinsPathologymedicine.medical_specialtyIschemia030218 nuclear medicine & medical imaging03 medical and health sciencesCerebral circulation0302 clinical medicineOcclusionLaser-Doppler FlowmetryAnimalsMedicineFluorescein AngiographyRats WistarCerebral perfusion pressureCerebral CortexBlood Volumebusiness.industryIntracranial Embolism and Thrombosismedicine.diseaseCortical VeinVein occlusionRatsNeurologyCerebral blood flowCerebrovascular CirculationNeurology (clinical)Cardiology and Cardiovascular Medicinebusiness030217 neurology & neurosurgeryJournal of Cerebral Blood Flow & Metabolism
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On the mechanism of action of phenylephrine in rat atrial heart muscle

1994

Both in rat left atrial heart and in aortic smooth muscle preparations, phenylephrine (PE) caused a concentration-dependent increase in force of contraction (FC) in the presence of atenolol (10 mumol/l), which was antagonized by phentolamine, prazosin and WB 4101 in a competitive manner. The pA2 values of the antagonists in the cardiac tissue were 10-20fold lower than those in the rat thoracic aorta. In the spontaneously beating right atrium, PE exerted a positive chronotropic action, which was not significantly antagonized by phentolamine or prazosin. It is therefore assumed that the effects of phenylephrine in the left atrium and in the aorta are mediated by different subtypes of alpha 1-…

MaleChronotropicmedicine.medical_specialtyPotassium ChannelsSodium-Hydrogen ExchangersAction PotentialsIn Vitro TechniquesRats Sprague-DawleyPhenylephrinePhentolamineHeart RateReceptors Adrenergic alpha-1medicine.arteryInternal medicinemedicinePrazosinAnimalsHeart AtriaPhenylephrineAdrenergic alpha-AntagonistsPharmacologyAortaChemistryCalcium RadioisotopesHeartGeneral MedicineAtenololMyocardial ContractionRatsElectrophysiologyActin CytoskeletonEndocrinologyMechanism of actioncardiovascular systemCalciummedicine.symptomAdrenergic alpha-Agonistsmedicine.drugMuscle contractionNaunyn-Schmiedeberg’s Archives of Pharmacology
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Calcium antagonist properties of cinnarizine, trifluoperazine and verapamil in guinea-pig normal and skinned trachealis muscle.

1990

In guinea-pig trachealis, depolarized by a K(+)-rich medium, Ca2+ (0.01-10 mM) caused concentration-related spasm. Verapamil (0.5-5 microM), cinnarizine (10-100 microM) and trifluoperazine (16-160 microM) each produced concentration-dependent antagonism of Ca2+ characterized by a rightward and downward displacement of the log concentration-effect curve for Ca2+. The rank order of potencies of these antagonists, measured as the IC75 against Ca2+ (10 mM)-induced contraction of depolarized trachea, was verapamil (5.6 microM) greater than cinnarizine (59 microM) greater than trifluoperazine (91 microM). In skinned trachea, verapamil in concentrations up to 100 microM did not modify the concentr…

MaleCinnarizineContraction (grammar)StereochemistryGuinea PigsPharmaceutical Sciencechemistry.chemical_elementTrifluoperazinePharmacologyCalciumIn Vitro TechniquesCinnarizinemedicineAnimalsPharmacologyAntagonistMuscle SmoothCalcium Channel BlockersTrifluoperazineTracheaMechanism of actionchemistryVerapamilTrachealis muscleVerapamilFemalemedicine.symptommedicine.drugThe Journal of pharmacy and pharmacology
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